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This Concept Map, created with IHMC CmapTools, has information related to: PEM, PKA phosphorylates and activates hormone sensitive lipase, decreased food intake causes a decrease in insulin, glucagon while glycogen stores are available, activates glycogenolysis, Marasmus type characterized by decreased food intake, gluconeogenesis in a starving patient there is insufficient substrate leading to decreased Fasting blood glucose levels, Urine creatinine excretion was below normal, 0.6 mmole/day, amino acids are transported and used for synthesis of serum proteins, 10 month old male patient presents with Muscle wasting, insulin a decrease in which does not activate protein phosphatase to dephosphorylate (activate) acetyl CoA carboxylase, skeletal muscle During starvation Ketone body metabolism, Acetyl CoA goes into TCA cycle, Malonyl CoA is the main substrate for fatty acid synthesis, high NADH levels drives synthesis of ketone bodies, acyl CoA dehydrogenase and other enzymes resulting in Fatty acyl CoA, amino acids when broken down release nitrogen products which are removed from the body via the Urea Cycle, decreased food intake occurs when calorie deprivation is greater than the reduction in protein, fatty acid synthesis is not occuring in this patient because of high levels of glucagon, glucose is no longer predominant energy source for Brain, 10 month old male patient laboratory tests included Fasting blood glucose levels, Serum albumin levels was below normal, 1.8 g/dL