work until it is reactivated.
This Concept Map, created with IHMC CmapTools, has information related to: Two Infants, glucose-6-Phosphatase deficiency ???? Build up of G6P, Lactate leading to Lactic acidemia, adipose tissue resulting in Latic Acid, gluconeogenesis inhibition results in Hypoglycemia, 20 mg/dL Hypoglycemia suggests glucose-6-Phosphatase deficiency, Build up of G6P occurs because of diffeciency with glucose-6- phosphatase, fructose 1,6 bisphosphatase regulates gluconeogenesis, Fructose is phyosphorylated by fructokinase, Fasting causes catabolism of adipose tissue, Lactose ???? Glycolysis, AMP degredation of results in Hyperuricemia, ATP/AMP Ratio when low will not stimulate gluconeogenesis, Hyperuricemia causes decreased excretion of Lactate, G-6-Phosphatase producing free Glucose, Baby 1 Presents Hyperuricemia, lactic acidemia suggests glucose-6-Phosphatase deficiency, G-6-P is dephosphorylated by G-6-Phosphatase, TCA cycle ???? Glycolysis, fructokinase and converted to Fructose 1-Phosphate, ATP/AMP Ratio when high stimulates fructose 1,6 bisphosphatase