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This Concept Map, created with IHMC CmapTools, has information related to: Tale of two infants, Inorganic Phosphate decreasing availability for Oxidative Phosphorylation, Fructose 1-P decreasing levels of Inorganic Phosphate, Glucose 6-P intermediate in Glycolysis, TCA cycle reducing activity of Oxidative Phosphorylation, Fructose phosphorylated by Fruktokinase to Fructose 1-P, cyclic AMP leading to activation of PKA, Baby 1 presents hyperuricemia, Aldolase B Deficiency which prevents formation of Glyceraldehyde, Gluconeogenesis leading to 20 mg/dl hypoglycemic, Baby 2 presents seizure, Baby 2 presents 20 mg/dl hypoglycemic, seizure suggests Glycogen-Storage Disease Type I, Glycolysis reducing activity of TCA cycle, Glucose 1,6-P then to Glucose 6-P, Glycolysis drastic increase of substrates overwhelms the enzymatic capacity of Citrate synthase, Glycogen by binding to a Gs protein- coupled receptor, activating Adenyl Cyclase, Glycogen causing bilaterally enlarged kidneys, Fructose 1-P Cleaved by Aldolase B to form Glyceraldehyde, Fructose 1,6 Bisphosphatase preventing cleavage of phosphate from Fructose 1,6-bis-P, Glucose 6-P Translocase into ER Glucose 6-P