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This Concept Map, created with IHMC CmapTools, has information related to: Neonatal Hypoglycemia, increased fetal insulin release will ultimately (due to its metabolic effects) increases fetal oxygen consumption, increased fetal insulin release due to increased insulin and low levels of glucagon and epinephrine leading to decreased gluconeogenesis, Muscle increased insulin release will decrease proteolysis, Pyruvate is normally converted to oxaloacetate, Infant Born with Enlarged Organs, increases fetal oxygen consumption leading to inadequate tissue oxygenation, ketogenesis decrease resulting in poor brain energy utalization and development, increased fetal blood glucose leads to decreased glucagon, catecholamines, and corticosteroid hormones, Liver increased size due to decreased Lipolysis, Enlarged Organs including enlarged Liver, inadequate tissue oxygenation causing baby's Hypoxia, Blood Glucose is an energy source for Other Tissues, increased fetal insulin release due to increased insulin and low levels of glucagon and epinephrine leading to decreased blood glucose levels, Skeletal Muscle Proteolysis when inhibited leads to decreased availability of amino acids, increased glucagon, catecholamines, and corticosteroid hormones; decreased insulin in the first couple days before feeding, this stimulates gluconeogenesis, proteolysis produces amino acids, glycogenolysis with active phosphorylase will catalyze the degradation of glycogen to G6P and the further metabolism of G6P, anaerobic glycolysis leading to a buildup of lactate, Beta oxidation in normal infant forms few ketone bodies, Macrosomia caused by increased (from the increased insulin lipogenesis